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Cellular Safety Shelters Allow TB Agent To Survive In Infected Individuals
Cellular Safety Shelters Allow TB Agent To Survive In Infected Individuals
Cellular Safety Shelters Allow TB Agent To Survive In Infected Individuals
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"Foamy" macrophage formation may be the key to persistence of infection by Mycobacterium tuberculosis (Mtb), the causative agent of tuberculosis, explains a new study in PLoS Pathogens. These immunity-related cells are shown to be a safety reservoir where the bacterium can hide for years in infected individuals, before inducing an active disease.
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Other Articles:
| • | Gentle Observation of Living Vells Over a Long Period of Time - AZoOptics.com (press release) | ||
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| • | Study looks at investigational compound MK-677 in Alzheimer's disease | ||
| A new study shows that a drug that increases the release of growth hormone failed to slow the rate of progression of Alzheimer's disease in humans. | |||
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| • | Cell: Highlights of 2006 | ||
| A look back at 2006, featuring interviews with two of this year's Nobel Laureates, Dr Craig Mello and Dr Roger Kornberg, as well as Dr Paul Nurse, president of New York's Rockefeller University, sharing his views on how the results of the recent midterm elections may influence biomedical research funding in the United States | |||
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| • | Untitled Document | ||
| Sickle Cell anemia is a life-threatening, inherited disorder, that affects the normal functioning of the red blood cells. | |||
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| • | Glucose metabolism by lymphocytes, macrophages, and tumor cells from Walker 256 tumor-bearing rats supplemented with fish oil for one generation | ||
| Here we investigated the effect of lifelong supplementation of the diet with coconut fat (CO, rich in saturated fatty acids) or fish oil (FO, rich in n-3 polyunsaturated fatty acids) on tumor growth and lactate production from glucose in Walker 256 tumor cells, peritoneal macrophages, spleen, and gut-associated lymphocytes. Female Wistar rats were supplemented with CO or FO prior to mating and then throughout pregnancy and gestation and then the male offspring were supplemented from weaning until 90 days of age. Then they were inoculated subcutaneously with Walker 256 tumor cells. Tumor weight at 14 days in control rats (those fed standard chow) and CO supplemented was approximately 30 g. Supplementation of the diet with FO significantly reduced tumor growth by 76%. Lactate production (nmol h-1 mg-1 protein) from glucose by Walker 256 cells in the group fed regular chow (W) was 381.8 ± 14.9. Supplementation with coconut fat (WCO) caused a significant reduction in lactate production by 1.6-fold and with fish oil (WFO) by 3.8-fold. Spleen lymphocytes obtained from W and WCO groups had markedly increased lactate production (553 ± 70 and 635 ± 150) when compared to non-tumor-bearing rats ([sim]260 ± 30). FO supplementation reduced significantly the lactate production (297 ± 50). Gut-associated lymphocytes obtained from W and WCO groups increased lactate production markedly (280 ± 31 and 276 ± 25) when compared to non-tumor-bearing rats ([sim]90 ± 18). FO supplementation reduced significantly the lactate production (168 ± 14). Lactate production by peritoneal macrophages was increased by tumor burden but there was no difference between the groups fed the various diets. Lifelong consumption of FO protects against tumor growth and modifies glucose metabolism in Walker tumor cells and lymphocytes but not in macrophages. Copyright © 2008 John Wiley & Sons, Ltd. | |||
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| • | New Triplantanol Search engine | ||
| New Triplantanol Search engine powered by Google added. Now you can better search our site or the web. Also Add to Google button placed on site. | |||
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ScienceDaily: Cell Biology News
By John Wiley & Sons, Inc - ©2008 Google - version: v1.5 build A
By John Wiley & Sons, Inc - ©2008 Google - version: v1.5 build A